What is in this article?:
- Similarities exist between resistant weeds and drugs
- Continued, widespread use is catalyst for resistance
- New products, management systems needed
Rather than always taking the extra time in a hectic schedule to educate the patient between the common cold and sinus infection, Wagner said a doctor or provider would commonly prescribe an antibiotic.
“Once antibiotic therapy was started, in many instances, the patient would start to feel better and would stop the antibiotic before it was all completed.”
That’s led to the latest challenge in overcoming a multi-antibiotic resistant staph aureus (bacterium) called MRSA.
“I see this in my clinic nearly every day,” Wagner said. “It’s very difficult to treat. When you eliminate penicillin, erythromycin and cephalosporin classes, you are limited in your antibiotic options. For those not allergic, we’ve resorted to using an older sulfa antibiotic to treat MRSA because of these limitations.”
Just like glyphosate, Wagner says the synthetic penicillin worked for a while, but over time “it couldn’t easily reach the site of activity.”
“The staph’s penicillin-binding protein lowered its affinity, creating a layer of protection,” Wagner said. “Thus, the penicillin antibiotic doesn’t fully penetrate.”
“It’s very analogous to the way herbicides work,” Baumann said. “The site of activity for an herbicide may be altered in resistant species, causing the herbicide to be ineffective. For example, in an acre, there may be one seed in millions that has an altered binding site. When you control 99 percent of all of the other targeted weed species, the genetic anomaly flourishes and all heck breaks loose.”